000 01499 a2200385 4500
005 20250514061833.0
264 0 _c20030421
008 200304s 0 0 eng d
022 _a0027-8424
024 7 _a10.1073/pnas.0634912100
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aRoca, Ramon
245 0 0 _aLoss of p16INK4a results in increased glucocorticoid receptor activity during fibrosarcoma development.
_h[electronic resource]
260 _bProceedings of the National Academy of Sciences of the United States of America
_cMar 2003
300 _a3113-8 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAnimals
650 0 4 _aBase Sequence
650 0 4 _aCell Division
_xdrug effects
650 0 4 _aCyclin-Dependent Kinase Inhibitor p16
_xdeficiency
650 0 4 _aDNA Methylation
650 0 4 _aDNA, Neoplasm
_xchemistry
650 0 4 _aDexamethasone
_xpharmacology
650 0 4 _aFibrosarcoma
_xetiology
650 0 4 _aGenes, p16
650 0 4 _aMice
650 0 4 _aPrecancerous Conditions
_xetiology
650 0 4 _aReceptors, Glucocorticoid
_xgenetics
650 0 4 _aTranscriptional Activation
650 0 4 _aTumor Cells, Cultured
700 1 _aKypta, Robert M
700 1 _aVivanco, Maria d M
773 0 _tProceedings of the National Academy of Sciences of the United States of America
_gvol. 100
_gno. 6
_gp. 3113-8
856 4 0 _uhttps://doi.org/10.1073/pnas.0634912100
_zAvailable from publisher's website
999 _c12419572
_d12419572