000 01637 a2200469 4500
005 20250514023333.0
264 0 _c20020305
008 200203s 0 0 eng d
022 _a0008-5472
040 _aNLM
_beng
_cNLM
100 1 _aCharbonnier, Françoise
245 0 0 _aMSH2 in contrast to MLH1 and MSH6 is frequently inactivated by exonic and promoter rearrangements in hereditary nonpolyposis colorectal cancer.
_h[electronic resource]
260 _bCancer research
_cFeb 2002
300 _a848-53 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAdaptor Proteins, Signal Transducing
650 0 4 _aCarrier Proteins
650 0 4 _aColorectal Neoplasms, Hereditary Nonpolyposis
_xgenetics
650 0 4 _aDNA-Binding Proteins
_xgenetics
650 0 4 _aExons
650 0 4 _aGene Expression Regulation, Neoplastic
650 0 4 _aGene Rearrangement
650 0 4 _aGene Silencing
650 0 4 _aHumans
650 0 4 _aMiddle Aged
650 0 4 _aMutL Protein Homolog 1
650 0 4 _aMutS Homolog 2 Protein
650 0 4 _aNeoplasm Proteins
_xgenetics
650 0 4 _aNuclear Proteins
650 0 4 _aPoint Mutation
650 0 4 _aPolymerase Chain Reaction
_xmethods
650 0 4 _aPromoter Regions, Genetic
_xgenetics
650 0 4 _aProto-Oncogene Proteins
_xgenetics
700 1 _aOlschwang, Sylviane
700 1 _aWang, Qing
700 1 _aBoisson, Cécile
700 1 _aMartin, Cosette
700 1 _aBuisine, Marie-Pierre
700 1 _aPuisieux, Alain
700 1 _aFrebourg, Thierry
773 0 _tCancer research
_gvol. 62
_gno. 3
_gp. 848-53
999 _c11739750
_d11739750