000 01580 a2200433 4500
005 20250513233234.0
264 0 _c20011204
008 200112s 0 0 eng d
022 _a0027-8424
024 7 _a10.1073/pnas.051378298
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aForcet, C
245 0 0 _aThe dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation.
_h[electronic resource]
260 _bProceedings of the National Academy of Sciences of the United States of America
_cMar 2001
300 _a3416-21 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
650 0 4 _aApoptosis
650 0 4 _aCaspase 3
650 0 4 _aCaspase 9
650 0 4 _aCaspases
_xmetabolism
650 0 4 _aCell Adhesion Molecules
_xgenetics
650 0 4 _aCell Line, Transformed
650 0 4 _aColorectal Neoplasms
650 0 4 _aDCC Receptor
650 0 4 _aEnzyme Activation
650 0 4 _aGenes, Tumor Suppressor
650 0 4 _aHumans
650 0 4 _aReceptors, Cell Surface
650 0 4 _aTumor Cells, Cultured
650 0 4 _aTumor Suppressor Proteins
_xgenetics
700 1 _aYe, X
700 1 _aGranger, L
700 1 _aCorset, V
700 1 _aShin, H
700 1 _aBredesen, D E
700 1 _aMehlen, P
773 0 _tProceedings of the National Academy of Sciences of the United States of America
_gvol. 98
_gno. 6
_gp. 3416-21
856 4 0 _uhttps://doi.org/10.1073/pnas.051378298
_zAvailable from publisher's website
999 _c11184968
_d11184968