| 000 | 01800 a2200529 4500 | ||
|---|---|---|---|
| 005 | 20250513192509.0 | ||
| 264 | 0 | _c19991007 | |
| 008 | 199910s 0 0 eng d | ||
| 022 | _a0002-9440 | ||
| 024 | 7 |
_a10.1016/S0002-9440(10)65167-X _2doi |
|
| 040 |
_aNLM _beng _cNLM |
||
| 100 | 1 | _aObici, L | |
| 245 | 0 | 0 |
_aThe new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide. _h[electronic resource] |
| 260 |
_bThe American journal of pathology _cSep 1999 |
||
| 300 |
_a695-702 p. _bdigital |
||
| 500 | _aPublication Type: Case Reports; Journal Article; Research Support, Non-U.S. Gov't | ||
| 650 | 0 | 4 | _aAmino Acid Substitution |
| 650 | 0 | 4 |
_aAmyloid _xchemistry |
| 650 | 0 | 4 |
_aAmyloidosis _xcomplications |
| 650 | 0 | 4 |
_aApolipoprotein A-I _xchemistry |
| 650 | 0 | 4 | _aChromatography, Gel |
| 650 | 0 | 4 |
_aHeart Diseases _xetiology |
| 650 | 0 | 4 | _aHumans |
| 650 | 0 | 4 | _aMale |
| 650 | 0 | 4 | _aMass Spectrometry |
| 650 | 0 | 4 | _aMiddle Aged |
| 650 | 0 | 4 | _aModels, Molecular |
| 650 | 0 | 4 | _aMolecular Weight |
| 650 | 0 | 4 |
_aMyocardium _xchemistry |
| 650 | 0 | 4 |
_aPeptide Fragments _xchemistry |
| 650 | 0 | 4 |
_aPoint Mutation _xgenetics |
| 650 | 0 | 4 | _aPolymerase Chain Reaction |
| 650 | 0 | 4 | _aSequence Analysis, DNA |
| 700 | 1 | _aBellotti, V | |
| 700 | 1 | _aMangione, P | |
| 700 | 1 | _aStoppini, M | |
| 700 | 1 | _aArbustini, E | |
| 700 | 1 | _aVerga, L | |
| 700 | 1 | _aZorzoli, I | |
| 700 | 1 | _aAnesi, E | |
| 700 | 1 | _aZanotti, G | |
| 700 | 1 | _aCampana, C | |
| 700 | 1 | _aViganò, M | |
| 700 | 1 | _aMerlini, G | |
| 773 | 0 |
_tThe American journal of pathology _gvol. 155 _gno. 3 _gp. 695-702 |
|
| 856 | 4 | 0 |
_uhttps://doi.org/10.1016/S0002-9440(10)65167-X _zAvailable from publisher's website |
| 999 |
_c10445149 _d10445149 |
||