Impaired neutrophil chemotaxis in sepsis associates with GRK expression and inhibition of actin assembly and tyrosine phosphorylation. [electronic resource]
Producer: 20061128Description: 2906-13 p. digitalISSN:- 0006-4971
- Actins -- biosynthesis
- Cell Movement
- Chemotaxis, Leukocyte -- physiology
- Cytokines -- pharmacology
- DNA Primers
- G-Protein-Coupled Receptor Kinase 2
- G-Protein-Coupled Receptor Kinase 5
- Humans
- Lipopolysaccharides -- pharmacology
- N-Formylmethionine Leucyl-Phenylalanine -- pharmacology
- Neutrophils -- drug effects
- Phosphotyrosine -- metabolism
- Protein Serine-Threonine Kinases -- genetics
- Receptors, Interleukin-8A -- genetics
- Reverse Transcriptase Polymerase Chain Reaction
- Sepsis -- blood
- beta-Adrenergic Receptor Kinases -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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