HCV induces oxidative and ER stress, and sensitizes infected cells to apoptosis in SCID/Alb-uPA mice. [electronic resource]
Producer: 20090505Description: e1000291 p. digitalISSN:- 1553-7374
- Animals
- Apoptosis
- Endoplasmic Reticulum -- physiology
- Endoplasmic Reticulum Chaperone BiP
- Gene Expression Profiling
- Gene Expression Regulation
- Heat-Shock Proteins -- metabolism
- Hepacivirus -- physiology
- Hepatitis C -- immunology
- Hepatitis C Antibodies -- metabolism
- Hepatocytes -- metabolism
- Humans
- Immunohistochemistry
- In Situ Nick-End Labeling
- Lipid Metabolism
- Liver -- metabolism
- Mice
- Mice, SCID
- Microscopy, Confocal
- Molecular Chaperones -- metabolism
- NF-kappa B -- metabolism
- Oxidative Stress
- Stress, Physiological
- bcl-2-Associated X Protein -- metabolism
- bcl-X Protein -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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