Endothelial damage effects of circulating microparticles from patients with stable angina are reduced by aspirin through ERK/p38 MAPKs pathways. [electronic resource]
Producer: 20180504ISSN:- 1755-5922
- Angina, Stable -- pathology
- Animals
- Aorta, Thoracic -- pathology
- Aspirin -- therapeutic use
- Cell-Derived Microparticles
- Endothelium, Vascular -- drug effects
- Healthy Volunteers
- Humans
- MAP Kinase Signaling System -- drug effects
- Male
- NF-kappa B -- metabolism
- Nitric Oxide -- biosynthesis
- Platelet Aggregation Inhibitors -- therapeutic use
- Rats
- Superoxides -- metabolism
- Vascular Cell Adhesion Molecule-1 -- biosynthesis
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Publication Type: Journal Article
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