Neisseria meningitidis can induce pro-inflammatory cytokine production via pathways independent from CD14 and toll-like receptor 4. [electronic resource]
Producer: 20030926Description: 411-7 p. digitalISSN:- 1148-5493
- Animals
- Antibodies, Monoclonal -- pharmacology
- Cytokines -- biosynthesis
- Escherichia coli -- pathogenicity
- Humans
- In Vitro Techniques
- Inflammation Mediators -- metabolism
- Interleukin-1 -- biosynthesis
- Leukocytes, Mononuclear -- drug effects
- Lipopolysaccharide Receptors -- metabolism
- Lipopolysaccharides -- toxicity
- Macrophages, Peritoneal -- drug effects
- Membrane Glycoproteins -- metabolism
- Mice
- Mice, Inbred C3H
- Mice, Inbred C57BL
- Neisseria meningitidis -- pathogenicity
- Receptors, Cell Surface -- metabolism
- Toll-Like Receptor 4
- Toll-Like Receptors
- Tumor Necrosis Factor-alpha -- biosynthesis
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Publication Type: Journal Article
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