Pharmacological blockade of ERG K(+) channels and Ca(2+) influx through store-operated channels exerts opposite effects on intracellular Ca(2+) oscillations in pituitary GH(3) cells. [electronic resource]
Producer: 20001109Description: 1115-28 p. digitalISSN:- 0026-895X
- Animals
- Anti-Arrhythmia Agents -- pharmacology
- Astemizole -- pharmacology
- Biological Transport -- drug effects
- Calcium -- metabolism
- Calcium Channel Blockers -- pharmacology
- Calcium Channels -- drug effects
- Calcium Channels, L-Type -- metabolism
- Cation Transport Proteins
- Cells, Cultured
- Cetirizine -- pharmacology
- Electrophysiology
- Ether-A-Go-Go Potassium Channels
- Histamine H1 Antagonists -- pharmacology
- Hydroxyzine -- pharmacology
- Large-Conductance Calcium-Activated Potassium Channels
- Nimodipine -- pharmacology
- Phenethylamines -- pharmacology
- Pituitary Gland -- cytology
- Potassium Channel Blockers
- Potassium Channels -- metabolism
- Potassium Channels, Calcium-Activated
- Potassium Channels, Voltage-Gated
- Rats
- Small-Conductance Calcium-Activated Potassium Channels
- Sulfonamides -- pharmacology
- Terfenadine -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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