Amyloid-beta peptide oligomers disrupt axonal transport through an NMDA receptor-dependent mechanism that is mediated by glycogen synthase kinase 3beta in primary cultured hippocampal neurons. [electronic resource]
Producer: 20100816Description: 9166-71 p. digitalISSN:- 1529-2401
- Amyloid beta-Peptides -- toxicity
- Animals
- Axonal Transport -- drug effects
- Brain-Derived Neurotrophic Factor -- genetics
- Cells, Cultured
- Drug Interactions
- Enzyme Inhibitors -- pharmacology
- Excitatory Amino Acid Antagonists -- pharmacology
- Gene Expression Regulation -- drug effects
- Glycogen Synthase Kinase 3 -- antagonists & inhibitors
- Glycogen Synthase Kinase 3 beta
- Green Fluorescent Proteins -- genetics
- Hippocampus -- cytology
- Luminescent Proteins -- genetics
- Mice
- Mutation -- genetics
- Neurons -- cytology
- Receptors, N-Methyl-D-Aspartate -- antagonists & inhibitors
- Transfection -- methods
- Tubulin -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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