Gli2 gene-environment interactions contribute to the etiological complexity of holoprosencephaly: evidence from a mouse model. [electronic resource]
Producer: 20171205Description: 1307-1315 p. digitalISSN:- 1754-8411
- Animals
- Body Patterning
- Brain -- abnormalities
- Disease Models, Animal
- Face -- abnormalities
- Fetus -- abnormalities
- Gene-Environment Interaction
- Hedgehog Proteins -- metabolism
- Heterozygote
- Holoprosencephaly -- embryology
- Ligands
- Loss of Function Mutation -- genetics
- Male
- Mice, Inbred C57BL
- Teratogens -- toxicity
- Zinc Finger Protein Gli2 -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
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