TGFβ regulates persistent neuroinflammation by controlling Th1 polarization and ROS production via monocyte-derived dendritic cells. [electronic resource]
Producer: 20180104Description: 1925-37 p. digitalISSN:- 1098-1136
- Amyotrophic Lateral Sclerosis -- genetics
- Animals
- Cell Polarity -- genetics
- Cohort Studies
- Cytokines -- genetics
- Dendritic Cells -- physiology
- Disease Models, Animal
- Encephalomyelitis, Autoimmune, Experimental -- genetics
- Gene Expression Regulation -- genetics
- Genotype
- Humans
- Leukocyte Common Antigens -- genetics
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Monocytes -- cytology
- Myelin-Oligodendrocyte Glycoprotein -- immunology
- NADPH Oxidase 2 -- genetics
- Polymorphism, Single Nucleotide -- genetics
- Reactive Oxygen Species -- metabolism
- Th1 Cells -- physiology
- Transforming Growth Factor beta -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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