Cigarette smoke combined with Toll-like receptor 3 signaling triggers exaggerated epithelial regulated upon activation, normal T-cell expressed and secreted/CCL5 expression in chronic rhinosinusitis. [electronic resource]
Producer: 20090123Description: 1145-1153.e3 p. digitalISSN:- 1097-6825
- Adult
- Aged
- Cells, Cultured
- Chemokine CCL5 -- biosynthesis
- Chemokine CCL7 -- biosynthesis
- Chronic Disease
- Complex Mixtures -- pharmacology
- Epithelial Cells -- metabolism
- Female
- Gene Expression Regulation -- drug effects
- Humans
- Immunochemistry
- Interferon-beta -- biosynthesis
- Interferons
- Interleukin-1beta -- biosynthesis
- Interleukin-6 -- biosynthesis
- Interleukin-8 -- biosynthesis
- Interleukins -- biosynthesis
- Lipopolysaccharides -- pharmacology
- Male
- Middle Aged
- Nasal Mucosa -- metabolism
- RNA, Double-Stranded -- pharmacology
- RNA, Messenger -- biosynthesis
- Rhinitis
- Sinusitis
- Tobacco Smoke Pollution -- adverse effects
- Toll-Like Receptor 3 -- agonists
- Tumor Necrosis Factor-alpha -- pharmacology
- beta-Defensins -- biosynthesis
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Publication Type: Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
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