Lack of BACE1 S-palmitoylation reduces amyloid burden and mitigates memory deficits in transgenic mouse models of Alzheimer's disease. [electronic resource]
Producer: 20180618Description: E9665-E9674 p. digitalISSN:- 1091-6490
- Alzheimer Disease -- metabolism
- Amyloid -- metabolism
- Amyloid Precursor Protein Secretases -- metabolism
- Amyloid beta-Peptides -- metabolism
- Amyloid beta-Protein Precursor -- metabolism
- Amyloidogenic Proteins -- metabolism
- Amyloidosis -- metabolism
- Animals
- Aspartic Acid Endopeptidases -- metabolism
- Axons -- metabolism
- Brain -- metabolism
- Disease Models, Animal
- Female
- Lipoylation -- physiology
- Male
- Memory Disorders -- metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Neurons -- metabolism
- Protein Processing, Post-Translational -- physiology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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