Brain activity mapping in Mecp2 mutant mice reveals functional deficits in forebrain circuits, including key nodes in the default mode network, that are reversed with ketamine treatment. [electronic resource]
Producer: 20130117Description: 13860-72 p. digitalISSN:- 1529-2401
- Animals
- Autonomic Nervous System -- physiopathology
- Cerebellum -- metabolism
- Disease Models, Animal
- Excitatory Amino Acid Antagonists -- pharmacology
- Female
- Gene Expression Regulation, Developmental
- Genes, fos
- Humans
- Ketamine -- pharmacology
- Male
- Methyl-CpG-Binding Protein 2 -- deficiency
- Mice
- Mice, Knockout
- Miniature Postsynaptic Potentials -- drug effects
- Nerve Net -- drug effects
- Nerve Tissue Proteins -- biosynthesis
- Organ Specificity
- Patch-Clamp Techniques
- Prosencephalon -- metabolism
- Proto-Oncogene Proteins c-fos -- biosynthesis
- Rett Syndrome -- genetics
- Sensory Gating -- drug effects
- Solitary Nucleus -- chemistry
- Synaptic Transmission -- drug effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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