APA

He H., Wang Y., Guo X., Ramchandani S., Ma J., Shen M., Garcia D. A., Deng Y., Multani A. S., You M. J. & Chang S. (20090114). Pot1b deletion and telomerase haploinsufficiency in mice initiate an ATR-dependent DNA damage response and elicit phenotypes resembling dyskeratosis congenita. : Molecular and cellular biology.

Chicago

He Hua, Wang Yang, Guo Xiaolan, Ramchandani Sonal, Ma Jin, Shen Mei-Feng, Garcia Dennis A, Deng Yibin, Multani Asha S, You Mingjian James and Chang Sandy. 20090114. Pot1b deletion and telomerase haploinsufficiency in mice initiate an ATR-dependent DNA damage response and elicit phenotypes resembling dyskeratosis congenita. : Molecular and cellular biology.

Harvard

He H., Wang Y., Guo X., Ramchandani S., Ma J., Shen M., Garcia D. A., Deng Y., Multani A. S., You M. J. and Chang S. (20090114). Pot1b deletion and telomerase haploinsufficiency in mice initiate an ATR-dependent DNA damage response and elicit phenotypes resembling dyskeratosis congenita. : Molecular and cellular biology.

MLA

He Hua, Wang Yang, Guo Xiaolan, Ramchandani Sonal, Ma Jin, Shen Mei-Feng, Garcia Dennis A, Deng Yibin, Multani Asha S, You Mingjian James and Chang Sandy. Pot1b deletion and telomerase haploinsufficiency in mice initiate an ATR-dependent DNA damage response and elicit phenotypes resembling dyskeratosis congenita. : Molecular and cellular biology. 20090114.