Depletion of CLL-associated patrolling monocytes and macrophages controls disease development and repairs immune dysfunction in vivo. [electronic resource]
Producer: 20160719Description: 570-9 p. digitalISSN:- 1476-5551
- Animals
- Antigens, Ly -- genetics
- B7-H1 Antigen -- genetics
- Chemokine CXCL9 -- genetics
- Clodronic Acid -- pharmacology
- Dendritic Cells -- drug effects
- Disease Models, Animal
- Gene Expression Regulation, Leukemic
- Humans
- Immunophenotyping
- Interleukin-10 -- genetics
- Leukemia, Lymphocytic, Chronic, B-Cell -- drug therapy
- Macrophages -- drug effects
- Mice
- Mice, Transgenic
- Monocytes -- drug effects
- Peritoneal Cavity -- pathology
- Phenotype
- Receptors, CCR2 -- genetics
- Signal Transduction
- Spleen -- drug effects
- T-Lymphocytes -- drug effects
- Tumor Necrosis Factor-alpha -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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