Pulmonary receptor for advanced glycation end-products promotes asthma pathogenesis through IL-33 and accumulation of group 2 innate lymphoid cells. [electronic resource]
Producer: 20151125Description: 747-756.e4 p. digitalISSN:- 1097-6825
- Allergens -- administration & dosage
- Alternaria -- chemistry
- Animals
- Antigens, Dermatophagoides -- administration & dosage
- Asthma -- chemically induced
- Bone Marrow -- immunology
- Cell Proliferation
- Gastrointestinal Tract -- immunology
- Gene Expression Regulation
- Immunity, Innate
- Interleukin-13 -- genetics
- Interleukin-33 -- genetics
- Interleukin-5 -- genetics
- Lung -- immunology
- Lymphocytes -- immunology
- Mice
- Organ Specificity
- Peritoneum -- immunology
- Pyroglyphidae -- chemistry
- Receptor for Advanced Glycation End Products -- genetics
- Signal Transduction
- Spleen -- immunology
- Transplantation Chimera
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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