ATM regulates cell fate choice upon p53 activation by modulating mitochondrial turnover and ROS levels. [electronic resource]
Producer: 20150921Description: 56-63 p. digitalISSN:- 1551-4005
- Apoptosis -- drug effects
- Ataxia Telangiectasia Mutated Proteins -- antagonists & inhibitors
- Autophagy -- drug effects
- BH3 Interacting Domain Death Agonist Protein -- antagonists & inhibitors
- Caspase 8 -- chemistry
- HCT116 Cells
- Heat-Shock Proteins -- metabolism
- Humans
- Imidazoles -- pharmacology
- Mitochondria -- metabolism
- Mitochondrial Turnover -- drug effects
- Morpholines -- toxicity
- Nuclear Proteins -- metabolism
- Piperazines -- pharmacology
- Proto-Oncogene Proteins c-bcl-2 -- antagonists & inhibitors
- Pyrones -- toxicity
- RNA Interference
- RNA, Small Interfering -- metabolism
- Reactive Oxygen Species -- metabolism
- Tumor Suppressor Protein p53 -- metabolism
- bcl-2-Associated X Protein -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.