The role of TLR4 and HO-1 in neuroinflammation after subarachnoid hemorrhage. [electronic resource]
Producer: 20210712Description: 549-556 p. digitalISSN:- 1097-4547
- Animals
- Antigens, CD -- metabolism
- Antigens, Differentiation, Myelomonocytic -- metabolism
- Encephalitis -- etiology
- HMGB1 Protein -- metabolism
- Heme Oxygenase (Decyclizing) -- metabolism
- Humans
- Mitogen-Activated Protein Kinases -- metabolism
- Receptors, Cell Surface -- metabolism
- Signal Transduction
- Subarachnoid Hemorrhage -- complications
- Toll-Like Receptor 4 -- metabolism
- CD163 Antigen
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
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