Phosphatidylinositol 3-kinase/Akt signaling controls endothelial cell sensitivity to Fas-mediated apoptosis via regulation of FLICE-inhibitory protein (FLIP). [electronic resource]
Producer: 20010823Description: 13-9 p. digitalISSN:- 1524-4571
- Androstadienes -- pharmacology
- Antibodies -- immunology
- Apoptosis
- CASP8 and FADD-Like Apoptosis Regulating Protein
- Carrier Proteins -- metabolism
- Caspase 8
- Caspase 9
- Caspases -- metabolism
- Cells, Cultured
- Down-Regulation
- Endothelial Growth Factors -- pharmacology
- Endothelium, Vascular -- drug effects
- Enzyme Inhibitors -- pharmacology
- Enzyme Precursors -- metabolism
- Humans
- Intracellular Signaling Peptides and Proteins
- Jurkat Cells
- Lymphokines -- pharmacology
- Phosphatidylinositol 3-Kinases -- physiology
- Phosphoinositide-3 Kinase Inhibitors
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- antagonists & inhibitors
- Proto-Oncogene Proteins c-akt
- Signal Transduction
- Tetracycline -- pharmacology
- Vascular Endothelial Growth Factor A
- Vascular Endothelial Growth Factors
- Wortmannin
- fas Receptor -- immunology
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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