Deacetylation of the herpes simplex virus type 1 latency-associated transcript (LAT) enhancer and a decrease in LAT abundance precede an increase in ICP0 transcriptional permissiveness at early times postexplant. [electronic resource]
Producer: 20060224Description: 2063-8 p. digitalISSN:- 0022-538X
- Acetylation
- Animals
- Enhancer Elements, Genetic
- Female
- Ganglia, Spinal -- virology
- Gene Expression Regulation, Viral
- Herpesvirus 1, Human -- genetics
- Histones -- metabolism
- Immediate-Early Proteins -- genetics
- Mice
- Promoter Regions, Genetic
- RNA, Untranslated -- analysis
- RNA, Viral -- analysis
- Transcription, Genetic
- Ubiquitin-Protein Ligases -- genetics
- Virus Latency -- physiology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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