Nitric oxide inhibits dystrophin proteolysis by coxsackieviral protease 2A through S-nitrosylation: A protective mechanism against enteroviral cardiomyopathy. [electronic resource]
Producer: 20001121Description: 2276-81 p. digitalISSN:- 1524-4539
- Animals
- Blotting, Western
- COS Cells
- Cardiomyopathy, Dilated -- metabolism
- Catalytic Domain -- drug effects
- Cysteine -- metabolism
- Cysteine Endopeptidases -- drug effects
- Densitometry
- Dystrophin -- metabolism
- Enterovirus Infections -- metabolism
- Enzyme Activation -- drug effects
- Enzyme Inhibitors -- pharmacology
- HeLa Cells
- Humans
- Hydrazines -- pharmacology
- Mice
- Nitric Oxide -- metabolism
- Nitric Oxide Donors -- pharmacology
- Nitrogen Oxides
- Penicillamine -- analogs & derivatives
- Peptide Hydrolases -- metabolism
- S-Nitroso-N-Acetylpenicillamine
- Spermine -- analogs & derivatives
- Sulfhydryl Compounds -- metabolism
- Viral Fusion Proteins -- genetics
- Viral Proteins
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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