NA+/H+ exchanger 1- and aquaporin-1-dependent hyperosmolarity changes decrease nitric oxide production and induce VCAM-1 expression in endothelial cells exposed to high glucose. [electronic resource]
Producer: 20101216Description: 755-65 p. digitalISSN:- 0394-6320
- Aorta, Thoracic -- cytology
- Aquaporin 1 -- metabolism
- Blotting, Western
- Cation Transport Proteins -- metabolism
- Endothelial Cells -- drug effects
- Flow Cytometry
- Glucose -- pharmacology
- Humans
- Mannitol -- pharmacology
- Nitric Oxide -- biosynthesis
- Osmolar Concentration
- Protein Kinase C -- metabolism
- Protein Kinase C beta
- RNA, Small Interfering -- genetics
- Sodium-Hydrogen Exchanger 1
- Sodium-Hydrogen Exchangers -- metabolism
- Vascular Cell Adhesion Molecule-1 -- biosynthesis
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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