APA

Chanoit G., Lee S., Xi J., Zhu M., McIntosh R. A., Mueller R. A., Norfleet E. A. & Xu Z. (20081016). Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta. : American journal of physiology. Heart and circulatory physiology.

Chicago

Chanoit Guillaume, Lee SungRyul, Xi Jinkun, Zhu Min, McIntosh Rachel A, Mueller Robert A, Norfleet Edward A and Xu Zhelong. 20081016. Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta. : American journal of physiology. Heart and circulatory physiology.

Harvard

Chanoit G., Lee S., Xi J., Zhu M., McIntosh R. A., Mueller R. A., Norfleet E. A. and Xu Z. (20081016). Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta. : American journal of physiology. Heart and circulatory physiology.

MLA

Chanoit Guillaume, Lee SungRyul, Xi Jinkun, Zhu Min, McIntosh Rachel A, Mueller Robert A, Norfleet Edward A and Xu Zhelong. Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta. : American journal of physiology. Heart and circulatory physiology. 20081016.