Spermidine-enhanced autophagic flux improves cardiac dysfunction following myocardial infarction by targeting the AMPK/mTOR signalling pathway. [electronic resource]
Producer: 20200828Description: 3126-3142 p. digitalISSN:- 1476-5381
- AMP-Activated Protein Kinases -- antagonists & inhibitors
- Angiotensin II -- pharmacology
- Animals
- Apoptosis -- drug effects
- Cardiotonic Agents -- pharmacology
- Cell Survival -- drug effects
- Cells, Cultured
- Dose-Response Relationship, Drug
- Male
- Myocardial Infarction -- drug therapy
- Myocytes, Cardiac -- drug effects
- Rats
- Rats, Sprague-Dawley
- Signal Transduction -- drug effects
- Spermidine -- pharmacology
- Structure-Activity Relationship
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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