Inhibition of p38 mitogen-activated protein kinase attenuates left ventricular dysfunction by mediating pro-inflammatory cardiac cytokine levels in a mouse model of diabetes mellitus. [electronic resource]
Producer: 20070326Description: 2507-13 p. digitalISSN:- 0012-186X
- Animals
- Body Weight
- Cytokines -- metabolism
- Diabetes Mellitus, Experimental -- enzymology
- Diabetic Angiopathies -- physiopathology
- Enzyme Inhibitors -- therapeutic use
- Heart -- anatomy & histology
- Imidazoles -- therapeutic use
- Inflammation -- physiopathology
- Male
- Mice
- Mice, Inbred C57BL
- Organ Size
- Phosphorylation
- Pyridines -- therapeutic use
- Systole -- physiology
- Ventricular Dysfunction, Left -- physiopathology
- Ventricular Function, Left
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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