The mutation Ser511Asn leads to N-glycosylation and increases the cleavage of high molecular weight kininogen in rats genetically susceptible to inflammation. [electronic resource]
Producer: 20031124Description: 2835-42 p. digitalISSN:- 0006-4971
- Animals
- Bradykinin -- metabolism
- CHO Cells
- Cricetinae
- DNA Restriction Enzymes -- pharmacology
- Dextran Sulfate -- pharmacology
- Escherichia coli -- metabolism
- Glycosylation
- Inflammation
- Kaolin -- metabolism
- Kininogens -- genetics
- Mutation
- Peptidoglycan -- pharmacology
- Point Mutation
- Polymerase Chain Reaction
- Polysaccharides -- pharmacology
- Protein Structure, Tertiary
- Rats
- Rats, Inbred Lew
- Species Specificity
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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