A novel mouse model for ataxia-telangiectasia with a N-terminal mutation displays a behavioral defect and a low incidence of lymphoma but no increased oxidative burden. [electronic resource]
Producer: 20160912Description: 6331-49 p. digitalISSN:- 1460-2083
- Animals
- Ataxia Telangiectasia -- enzymology
- Ataxia Telangiectasia Mutated Proteins -- genetics
- Behavior, Animal -- physiology
- Cell Cycle Proteins -- genetics
- DNA Damage
- DNA-Binding Proteins -- genetics
- Disease Models, Animal
- Female
- Genetic Association Studies
- Humans
- Incidence
- Lymphoma, T-Cell -- enzymology
- Male
- Mice
- Mice, Inbred C57BL
- Mutation
- Oxidation-Reduction
- Tumor Suppressor Proteins -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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