Mutant and wild-type p53 form complexes with p73 upon phosphorylation by the kinase JNK. [electronic resource]
Producer: 20190916ISSN:- 1937-9145
- Apoptosis
- Apoptosis Regulatory Proteins -- genetics
- Binding Sites -- genetics
- Cell Line, Tumor
- Cell Survival
- Humans
- JNK Mitogen-Activated Protein Kinases -- chemistry
- Models, Molecular
- Mutation
- Phosphorylation
- Protein Binding
- Protein Conformation
- Signal Transduction
- Tumor Protein p73 -- chemistry
- Tumor Suppressor Protein p53 -- chemistry
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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