Cathepsin B-Deficient Mice Resolve Leishmania major Inflammation Faster in a T Cell-Dependent Manner. [electronic resource]
Producer: 20170330Description: e0004716 p. digitalISSN:- 1935-2735
- Adoptive Transfer
- Animals
- Antigen Presentation
- CD3 Complex -- analysis
- Cathepsin B -- deficiency
- Cathepsin L -- deficiency
- Cathepsins -- deficiency
- Dendritic Cells -- immunology
- Endopeptidases -- deficiency
- Foot
- Inflammation -- immunology
- Interferon-gamma -- biosynthesis
- Leishmania major -- immunology
- Leishmaniasis, Cutaneous -- immunology
- Macrophages -- immunology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Parasite Load
- Signal Transduction
- T-Lymphocyte Subsets -- immunology
- Th1 Cells -- immunology
- Toll-Like Receptor 9 -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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