Intersection of mechanisms of type 2A VWD through defects in VWF multimerization, secretion, ADAMTS-13 susceptibility, and regulated storage. [electronic resource]
Producer: 20120716Description: 4543-53 p. digitalISSN:- 1528-0020
- ADAM Proteins -- metabolism
- ADAMTS13 Protein
- Family
- Female
- HEK293 Cells
- Humans
- Male
- Mutation, Missense
- Protein Multimerization -- genetics
- Protein Processing, Post-Translational -- genetics
- Protein Transport -- genetics
- Proteolysis
- Signal Transduction -- genetics
- Transfection
- von Willebrand Disease, Type 2 -- genetics
- von Willebrand Factor -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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