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Genetic disruption of Kir6.2, the pore-forming subunit of ATP-sensitive K+ channel, predisposes to catecholamine-induced ventricular dysrhythmia. [electronic resource] by
- Liu, Xiao-Ke
- Yamada, Satsuki
- Kane, Garvan C
- Alekseev, Alexey E
- Hodgson, Denice M
- O'Cochlain, Fearghas
- Jahangir, Arshad
- Miki, Takashi
- Seino, Susumu
- Terzic, Andre
Producer: 20050414
In:
Diabetes vol. 53 Suppl 3
Availability: No items available.
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KCNJ11 gene knockout of the Kir6.2 KATP channel causes maladaptive remodeling and heart failure in hypertension. [electronic resource] by
- Kane, Garvan C
- Behfar, Atta
- Dyer, Roy B
- O'Cochlain, D Fearghas
- Liu, Xiao-Ke
- Hodgson, Denice M
- Reyes, Santiago
- Miki, Takashi
- Seino, Susumu
- Terzic, Andre
Producer: 20061017
In:
Human molecular genetics vol. 15
Availability: No items available.
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14.
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Gene knockout of the KCNJ8-encoded Kir6.1 K(ATP) channel imparts fatal susceptibility to endotoxemia. [electronic resource] by
- Kane, Garvan C
- Lam, Chen-Fuh
- O'Cochlain, Fearghas
- Hodgson, Denice M
- Reyes, Santiago
- Liu, Xiao-Ke
- Miki, Takashi
- Seino, Susumu
- Katusic, Zvonimir S
- Terzic, Andre
Producer: 20061127
In:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology vol. 20
Availability: No items available.
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15.
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Cardiopoietic programming of embryonic stem cells for tumor-free heart repair. [electronic resource] by
- Behfar, Atta
- Perez-Terzic, Carmen
- Faustino, Randolph S
- Arrell, D Kent
- Hodgson, Denice M
- Yamada, Satsuki
- Puceat, Michel
- Niederländer, Nicolas
- Alekseev, Alexey E
- Zingman, Leonid V
- Terzic, Andre
Producer: 20070326
In:
The Journal of experimental medicine vol. 204
Availability: No items available.
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16.
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Commitment of embryonic stem cells toward a cardiac lineage: molecular mechanisms and evidence for a promising therapeutic approach for heart failure. [electronic resource] by
- Méry, Annabelle
- Papadimou, Evangelia
- Zeineddine, Dana
- Ménard, Claudine
- Behfar, Atta
- Zingman, Leonid V
- Hodgson, Denice M
- Rauzier, Jean-Michel
- Kane, Garvan C
- Perez-Terzic, Carmen
- Terzic, André
- Pucéat, Michel
Producer: 20040628
In:
Journal of muscle research and cell motility vol. 24
Availability: No items available.
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17.
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Cellular remodeling in heart failure disrupts K(ATP) channel-dependent stress tolerance. [electronic resource] by
- Hodgson, Denice M
- Zingman, Leonid V
- Kane, Garvan C
- Perez-Terzic, Carmen
- Bienengraeber, Martin
- Ozcan, Cevher
- Gumina, Richard J
- Pucar, Darko
- O'Coclain, Fergus
- Mann, Douglas L
- Alekseev, Alexey E
- Terzic, Andre
Producer: 20030616
In:
The EMBO journal vol. 22
Availability: No items available.
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18.
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Transgenic overexpression of human DMPK accumulates into hypertrophic cardiomyopathy, myotonic myopathy and hypotension traits of myotonic dystrophy. [electronic resource] by
- O'Cochlain, D Fearghas
- Perez-Terzic, Carmen
- Reyes, Santiago
- Kane, Garvan C
- Behfar, Atta
- Hodgson, Denice M
- Strommen, Jeffrey A
- Liu, Xiao-Ke
- van den Broek, Walther
- Wansink, Derick G
- Wieringa, Bé
- Terzic, Andre
Producer: 20050425
In:
Human molecular genetics vol. 13
Availability: No items available.
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19.
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Kir6.2 is required for adaptation to stress. [electronic resource] by
- Zingman, Leonid V
- Hodgson, Denice M
- Bast, Peter H
- Kane, Garvan C
- Perez-Terzic, Carmen
- Gumina, Richard J
- Pucar, Darko
- Bienengraeber, Martin
- Dzeja, Petras P
- Miki, Takashi
- Seino, Susumu
- Alekseev, Alexey E
- Terzic, Andre
Producer: 20021113
In:
Proceedings of the National Academy of Sciences of the United States of America vol. 99
Availability: No items available.
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20.
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ABCC9 mutations identified in human dilated cardiomyopathy disrupt catalytic KATP channel gating. [electronic resource] by
- Bienengraeber, Martin
- Olson, Timothy M
- Selivanov, Vitaliy A
- Kathmann, Eva C
- O'Cochlain, Fearghas
- Gao, Fan
- Karger, Amy B
- Ballew, Jeffrey D
- Hodgson, Denice M
- Zingman, Leonid V
- Pang, Yuan-Ping
- Alekseev, Alexey E
- Terzic, Andre
Producer: 20040816
In:
Nature genetics vol. 36
Availability: No items available.
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