Resistance to TRAIL in non-transformed cells is due to multiple redundant pathways. [electronic resource]
Producer: 20140225Description: e702 p. digitalISSN:- 2041-4889
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- CASP8 and FADD-Like Apoptosis Regulating Protein -- metabolism
- Caspase 3 -- metabolism
- Cells, Cultured
- Cycloheximide -- pharmacology
- Drug Resistance, Neoplasm
- Fibroblasts -- drug effects
- GPI-Linked Proteins -- metabolism
- Humans
- Myocytes, Smooth Muscle -- drug effects
- Protein Synthesis Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Receptors, Tumor Necrosis Factor, Member 10c
- Signal Transduction
- Skin -- cytology
- TNF-Related Apoptosis-Inducing Ligand -- pharmacology
- Tumor Necrosis Factor Decoy Receptors -- metabolism
- X-Linked Inhibitor of Apoptosis Protein -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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