A CCL1/CCR8-dependent feed-forward mechanism drives ILC2 functions in type 2-mediated inflammation. [electronic resource]
Producer: 20200615Description: 2763-2777 p. digitalISSN:- 1540-9538
- Animals
- Autocrine Communication
- Biomarkers
- Cell Movement -- genetics
- Chemokine CCL1 -- metabolism
- Cytokines -- metabolism
- Gene Expression
- Helminths -- immunology
- Host-Parasite Interactions -- genetics
- Humans
- Immunity, Innate
- Immunophenotyping
- Inflammation -- etiology
- Inflammation Mediators -- metabolism
- Lymphocyte Subsets -- immunology
- Mice
- Mice, Knockout
- Receptors, CCR8 -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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