Spinal mediators that may contribute selectively to antinociceptive tolerance but not other effects of morphine as revealed by deletion of GluR5. [electronic resource]
Producer: 20110304Description: 475-87 p. digitalISSN:- 1873-7544
- Animals
- Calcitonin Gene-Related Peptide -- biosynthesis
- Drug Implants
- Drug Tolerance -- physiology
- Dynorphins -- biosynthesis
- Enzyme Activation -- drug effects
- Enzyme Induction -- drug effects
- Exploratory Behavior -- drug effects
- Male
- Mice
- Mice, Knockout
- Mitogen-Activated Protein Kinase 1 -- biosynthesis
- Morphine -- administration & dosage
- Morphine Dependence -- physiopathology
- Narcotics -- administration & dosage
- Pain -- drug therapy
- Pain Threshold -- drug effects
- Phosphorylation
- Posterior Horn Cells -- drug effects
- Protein Kinase C -- biosynthesis
- Protein Processing, Post-Translational
- Receptors, Kainic Acid -- deficiency
- p38 Mitogen-Activated Protein Kinases -- biosynthesis
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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