Nonsteroidal anti-inflammatory drugs inhibit re-epithelialization of wounded gastric monolayers by interfering with actin, Src, FAK, and tensin signaling. [electronic resource]
Producer: 20011228Description: 3055-71 p. digitalISSN:- 0024-3205
- Actins -- metabolism
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Cell Line
- Cell Movement -- drug effects
- Cyclooxygenase Inhibitors -- pharmacology
- Cytoskeleton -- drug effects
- Dose-Response Relationship, Drug
- Drug Interactions
- Epidermal Growth Factor -- pharmacology
- Focal Adhesion Kinase 1
- Focal Adhesion Protein-Tyrosine Kinases
- Gastric Mucosa -- drug effects
- Indomethacin -- pharmacology
- Microfilament Proteins -- metabolism
- Nitrobenzenes -- pharmacology
- Protein-Tyrosine Kinases -- metabolism
- Rats
- Signal Transduction -- drug effects
- Sulfonamides -- pharmacology
- Tensins
- Wound Healing -- drug effects
- src-Family Kinases -- metabolism
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Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
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