Thrombin-activatable fibrinolysis inhibitor influences disease severity in humans and mice with pneumococcal meningitis. [electronic resource]
Producer: 20160916Description: 2076-86 p. digitalISSN:- 1538-7836
- Adult
- Aged
- Animals
- Brain Damage, Chronic -- etiology
- Carboxypeptidase B2 -- cerebrospinal fluid
- Cerebral Hemorrhage -- etiology
- Community-Acquired Infections -- blood
- Complement C3a -- cerebrospinal fluid
- Complement C3b -- cerebrospinal fluid
- Complement Membrane Attack Complex -- cerebrospinal fluid
- Cytokines -- blood
- Female
- Fibrinolysis
- Humans
- Male
- Meningitis, Meningococcal -- blood
- Meningitis, Pneumococcal -- blood
- Mice
- Mice, Inbred C57BL
- Middle Aged
- Polymorphism, Single Nucleotide
- Respiratory Insufficiency -- etiology
- Shock, Septic -- etiology
- Treatment Outcome
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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