Deletion of the receptor for advanced glycation end products reduces glomerulosclerosis and preserves renal function in the diabetic OVE26 mouse. [electronic resource]
Producer: 20100913Description: 2043-54 p. digitalISSN:- 1939-327X
- Animals
- Blood Glucose -- metabolism
- Chickens
- Diabetic Nephropathies -- genetics
- Disease Models, Animal
- Gene Deletion
- Glomerular Mesangium -- metabolism
- Glycated Hemoglobin -- metabolism
- Kidney -- metabolism
- Mice
- Mice, Inbred Strains
- Pyruvaldehyde -- metabolism
- Receptor for Advanced Glycation End Products
- Receptors, Immunologic -- deficiency
- Renal Insufficiency -- genetics
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Publication Type: Journal Article
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