Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome. [electronic resource]
Producer: 20200922Description: e13286 p. digitalISSN:- 1748-1716
- Amiloride -- administration & dosage
- Animals
- Dose-Response Relationship, Drug
- Epithelial Sodium Channel Blockers -- administration & dosage
- Epithelial Sodium Channels -- genetics
- Gene Expression Regulation -- drug effects
- Ion Channel Gating
- Mice
- Mice, Knockout
- Nephrotic Syndrome
- Oocytes
- Sodium -- metabolism
- Urokinase-Type Plasminogen Activator -- genetics
- Xenopus laevis
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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