Absence of FcγRIII results in increased proinflammatory response in FcγRIII-KO cardiac recipients. [electronic resource]
Producer: 20131202Description: 601-8 p. digitalISSN:- 1534-6080
- Animals
- Apoptosis
- Complement Activation
- Cytokines -- metabolism
- Graft Rejection -- blood
- Graft Survival
- Heart Transplantation -- adverse effects
- Inflammation -- blood
- Inflammation Mediators -- blood
- Isoantibodies -- blood
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocardium -- immunology
- Receptors, IgG -- deficiency
- Serum Amyloid P-Component -- metabolism
- Time Factors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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