Brain-derived neurotrophic factor activation of CaM-kinase kinase via transient receptor potential canonical channels induces the translation and synaptic incorporation of GluA1-containing calcium-permeable AMPA receptors. [electronic resource]
Producer: 20120924Description: 8127-37 p. digitalISSN:- 1529-2401
- 2-Amino-5-phosphonovalerate -- pharmacology
- Adamantane -- analogs & derivatives
- Animals
- Benzimidazoles -- pharmacology
- Brain-Derived Neurotrophic Factor -- physiology
- Calcium Channel Blockers -- pharmacology
- Calcium-Calmodulin-Dependent Protein Kinase Kinase -- antagonists & inhibitors
- Enzyme Inhibitors -- pharmacology
- Excitatory Amino Acid Antagonists -- pharmacology
- Female
- Gene Knockdown Techniques
- Hippocampus -- drug effects
- Imidazoles -- pharmacology
- Male
- Miniature Postsynaptic Potentials -- drug effects
- Naphthalimides -- pharmacology
- Neurons -- drug effects
- Primary Cell Culture
- RNA, Small Interfering -- administration & dosage
- Rats
- Rats, Sprague-Dawley
- Receptors, AMPA -- metabolism
- TOR Serine-Threonine Kinases -- metabolism
- TRPC Cation Channels -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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