Myeloid cell leukemia 1 is phosphorylated through two distinct pathways, one associated with extracellular signal-regulated kinase activation and the other with G2/M accumulation or protein phosphatase 1/2A inhibition. [electronic resource]
Producer: 20000816Description: 21688-94 p. digitalISSN:- 0021-9258
- Burkitt Lymphoma
- Cell Cycle -- physiology
- Cell Survival -- drug effects
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- G2 Phase
- Humans
- Marine Toxins
- Microtubules -- drug effects
- Mitogen-Activated Protein Kinases -- metabolism
- Mitosis
- Myeloid Cell Leukemia Sequence 1 Protein
- Neoplasm Proteins -- isolation & purification
- Nocodazole -- pharmacology
- Okadaic Acid -- pharmacology
- Oxazoles -- pharmacology
- Paclitaxel -- pharmacology
- Phosphorylation
- Protein Kinase C -- metabolism
- Protein Phosphatase 1
- Protein Tyrosine Phosphatases -- antagonists & inhibitors
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Tetradecanoylphorbol Acetate -- pharmacology
- Tumor Cells, Cultured
- Vinblastine -- pharmacology
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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