Rapid turnover of the mTOR complex 1 (mTORC1) repressor REDD1 and activation of mTORC1 signaling following inhibition of protein synthesis. [electronic resource]
Producer: 20080331Description: 3465-3475 p. digitalISSN:- 0021-9258
- Animals
- Cell Cycle Proteins -- metabolism
- Cycloheximide -- pharmacology
- Gene Expression Regulation
- Mice
- Mice, Transgenic
- Models, Biological
- Monomeric GTP-Binding Proteins -- metabolism
- Neuropeptides -- metabolism
- Phosphoproteins -- metabolism
- Protein Biosynthesis
- Protein Kinases -- metabolism
- Protein Synthesis Inhibitors -- pharmacology
- RNA, Messenger -- metabolism
- Ras Homolog Enriched in Brain Protein
- Signal Transduction
- TOR Serine-Threonine Kinases
- Transcription Factors -- physiology
- Tuberous Sclerosis Complex 2 Protein
- Tumor Suppressor Proteins
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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