Null mutations for exon III and exon IV of the p75 neurotrophin receptor gene enhance sympathetic sprouting in response to elevated levels of nerve growth factor in transgenic mice. [electronic resource]
Producer: 20060609Description: 416-26 p. digitalISSN:- 0014-4886
- Animals
- Cell Count -- methods
- Cell Enlargement
- Cerebellum -- cytology
- Enzyme-Linked Immunosorbent Assay -- methods
- Exons -- genetics
- Immunohistochemistry -- methods
- Mice
- Mice, Knockout
- Mutation
- Nerve Growth Factor -- metabolism
- Neurites -- physiology
- Neurons -- cytology
- Neuropeptide Y -- metabolism
- Receptor, Nerve Growth Factor -- deficiency
- Receptor, trkA -- metabolism
- Superior Cervical Ganglion -- cytology
- Sympathetic Nervous System -- cytology
- Trigeminal Ganglion -- cytology
- Tyrosine 3-Monooxygenase -- metabolism
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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