A critical role for transcription factor Smad4 in T cell function that is independent of transforming growth factor β receptor signaling. [electronic resource]
Producer: 20150416Description: 68-79 p. digitalISSN:- 1097-4180
- Animals
- Autoimmunity -- genetics
- Cell Proliferation -- genetics
- Cells, Cultured
- Graft vs Host Disease -- immunology
- Immune Tolerance -- genetics
- Lymphocyte Activation -- genetics
- Mice
- Mice, Inbred BALB C
- Mice, Inbred C57BL
- Mice, Knockout
- Proto-Oncogene Proteins c-myc -- metabolism
- Receptors, Transforming Growth Factor beta -- genetics
- Signal Transduction -- genetics
- Smad4 Protein -- genetics
- T-Lymphocyte Subsets -- physiology
- T-Lymphocytes, Regulatory -- physiology
- Transplantation Chimera
- Tumor Escape
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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