Oxidation of HRas cysteine thiols by metabolic stress prevents palmitoylation in vivo and contributes to endothelial cell apoptosis. [electronic resource]
Producer: 20120409Description: 832-41 p. digitalISSN:- 1530-6860
- Animals
- Apoptosis -- physiology
- Cattle
- Cells, Cultured
- Cysteine -- chemistry
- Diet, High-Fat -- adverse effects
- Dietary Carbohydrates -- administration & dosage
- Endothelial Cells -- cytology
- Glucose -- administration & dosage
- Lipoylation
- Mice
- Mice, Inbred C57BL
- Mitogen-Activated Protein Kinase Kinases -- antagonists & inhibitors
- Mutant Proteins -- chemistry
- Oxidation-Reduction
- Proto-Oncogene Proteins p21(ras) -- chemistry
- Signal Transduction
- Stress, Physiological
- Sucrose -- administration & dosage
- Superoxide Dismutase -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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