Tumor-derived mutations within the DNA-binding domain of p53 that phenotypically resemble the deletion of the proline-rich domain. [electronic resource]
Producer: 20000608Description: 1834-42 p. digitalISSN:- 0950-9232
- Animals
- Apoptosis
- Binding Sites
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclins -- genetics
- DNA -- metabolism
- DNA-Binding Proteins -- metabolism
- Genes, Tumor Suppressor
- Humans
- Nuclear Proteins -- metabolism
- Phenotype
- Phosphorylation
- Proline -- genetics
- Promoter Regions, Genetic
- Sequence Deletion
- Transcriptional Activation
- Tumor Cells, Cultured
- Tumor Protein p73
- Tumor Suppressor Protein p53 -- genetics
- Tumor Suppressor Proteins
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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