ATR-p53 restricts homologous recombination in response to replicative stress but does not limit DNA interstrand crosslink repair in lung cancer cells. [electronic resource]
Producer: 20120215Description: e23053 p. digitalISSN:- 1932-6203
- Animals
- Ataxia Telangiectasia Mutated Proteins
- Cell Cycle Proteins -- genetics
- Cell Line, Tumor
- Cell Survival -- drug effects
- Cells, Cultured
- Cross-Linking Reagents -- pharmacology
- DNA Breaks, Double-Stranded -- drug effects
- DNA Damage
- DNA Repair
- DNA Replication -- drug effects
- Flow Cytometry
- Homologous Recombination
- Humans
- Lung Neoplasms -- genetics
- Mice
- Mitomycin -- pharmacology
- Mutation
- Protein Serine-Threonine Kinases -- genetics
- RNA Interference
- Rad51 Recombinase -- genetics
- Thymidine -- pharmacology
- Transcriptional Activation -- genetics
- Tumor Suppressor Protein p53 -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
There are no comments on this title.
Log in to your account to post a comment.