The allosteric potentiation of nicotinic acetylcholine receptors by galantamine ameliorates the cognitive dysfunction in beta amyloid25-35 i.c.v.-injected mice: involvement of dopaminergic systems. [electronic resource]
Producer: 20070727Description: 1261-71 p. digitalISSN:- 0893-133X
- Amyloid beta-Peptides -- administration & dosage
- Animals
- Cognition Disorders -- chemically induced
- Conditioning, Psychological -- physiology
- Cues
- Dopamine -- metabolism
- Extracellular Space -- metabolism
- Fear -- drug effects
- Galantamine -- pharmacology
- Hippocampus -- drug effects
- Injections, Intraventricular
- Male
- Memory -- drug effects
- Mice
- Mice, Inbred ICR
- Muscarinic Antagonists -- pharmacology
- Peptide Fragments -- administration & dosage
- Receptors, Nicotinic -- drug effects
- Recognition, Psychology -- drug effects
- Scopolamine -- pharmacology
- Synaptic Transmission -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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